Basenji puppy

I pleased to announce the arrival of Lyra's litter born 24 May 2018 and Sierra's litter born 27 May 2018.

All puppies are spoken for.

Meet the Parents

Carefully selected for temperament, health, and conformation

Ch Tambuzi Seven Steams Of Soma

Ch Tambuzi Seven Steams Of Soma

River

River attained his championship title at 8.5 months. He has been a very consistent winner for best of breed, class in group and class in show awards. He has a superb temperament.

HEALTH INFORMATION
Fanconi Syndrome: Normal by Parentage History
Pyruvate Kinase Deficiency: Normal by Parentage History
Late Onset Progressive Retinal Atrophy: Normal by Parentage History
PennHIP: DI: Right = 0.50 Left 0.53. Breed average DI 0.46.
PPM: R: Fine iris to cornea + opacity L: Normal
Coloboma: Normal
PRA: Normal
Cataracts: Normal

MBIS Supreme Ch Tambuzi The Secret Society

MBIS Supreme Ch Tambuzi The Secret Society

Lyra

Lyra is forever the playful girl. She has done extremely well in the show ring and quickly attained the title of supreme champion. Lyra is my first homebred All Breeds Best in Show winner. She is now a multi All Breeds Best in Show and Multi Runner Up Best in Speciality Show winner.

HEALTH INFORMATION
Fanconi Syndrome: Normal by Parentage History
Pyruvate Kinase Deficiency: Normal by Parentage History
Late Onset Progressive Retinal Atrophy: Normal by Parentage History
PennHIP: DI: Right = 0.41 Left 0.37. Breed average DI 0.46.
PPM: Normal.
Coloboma: Normal
PRA: Normal
Cataracts: Normal

The family/pedigree

Fanconi Syndrome - Normal 100%
Fanconi Syndrome - Carrier 0%
Fanconi Syndrome - Affected 0%
Pyruvate Kinase Deficiency - Normal 100%
Pyruvate Kinase Deficiency - Carrier 0%
Pyruvate Kinase Deficiency - Affected 0%
PRA - BJ1 - Normal 50%
PRA - BJ1 - Carrier 50%
PRA - BJ1 - Affected 0%

Puppies from this litter will be normal (clear) for Fanconi Syndrome and Pyruvate Kinase Deficiency. Based on probability 50% of the litter may be normal (clear) of PRA-BJ1 and 50% may carry the disease. However, these percentages are a guide only, 100% may be normal (clear) or 100% may carry the disease. It should be noted that carriers only 'carry' one copy of the mutant gene responsible for this disease. Therefore, they will not actually become afflicted by it.

Ch Tambuzi The Master Of Ascallon

Ch Tambuzi The Master Of Ascallon

Gus

Gus resides with Karen, two Basenjis a Min Pin and a Chihuahua.He is very handsome and attained his championship title quickly even though I am unable to show him regularly.

HEALTH INFORMATION
Fanconi Syndrome: Normal by Parentage History
Pyruvate Kinase Deficiency: Normal by Parentage History
Late Onset Progressive Retinal Atrophy: Normal
PennHIP DI: Right = 0.42 Left 0.35. Breed average DI 0.46.
PPM: Normal.
Coloboma: Left eye: Tiny optic nerve head.
PRA: Normal
Cataracts:Normal

Ch Tambuzi How Soon Is Now

Ch Tambuzi How Soon Is Now

Sierra

Sierra is wickedly humorous. For a 7.5-year-old she hasn’t yet lost her puppy behaviour. Always gives her little dance routine before meal time. She is friendly and likes to meet new people. She is an excellent ear cleaner for dogs and humans and loves her kisses!

HEALTH INFORMATION
Fanconi Syndrome: Normal by Parentage History
Pyruvate Kinase Deficiency: Normal by Parentage History
Late Onset Progressive Retinal Atrophy: Carrier
AVA Hips: Right=3 Left=2. Score 5 Breed average 6.9.
PPM: Normal.
Coloboma: Normal
PRA: Normal
Cataracts: Normal

The family/pedigree

Fanconi Syndrome - Normal 100%
Fanconi Syndrome - Carrier 0%
Fanconi Syndrome - Affected 0%
Pyruvate Kinase Deficiency - Normal 100%
Pyruvate Kinase Deficiency - Carrier 0%
Pyruvate Kinase Deficiency - Affected 0%
PRA - BJ1 - Normal 50%
PRA - BJ1 - Carrier 50%
PRA - BJ1 - Affected 0%

Puppies from this litter will be normal (clear) for Fanconi Syndrome and Pyruvate Kinase Deficiency. Based on probability 50% of the litter may be normal (clear) of PRA-BJ1 and 50% may carry the disease. However, these percentages are a guide only, 100% may be normal (clear) or 100% may carry the disease. It should be noted that carriers only 'carry' one copy of the mutant gene responsible for this disease. Therefore, they will not actually become afflicted by it.